SBP3500511
IDH1 Blocking Peptide for SAB3500511
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10 MG
₹8,789.00
50 MG
₹34,705.00
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10 MG
₹8,789.00
50 MG
₹34,705.00
About This Item
UNSPSC Code:
12352203
NACRES:
NA.41
₹8,789.00
Please contact Customer Service for Availability
New, lower price on this item!
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biological source
synthetic
Quality Level
form
liquid
species reactivity
rat, mouse, human
concentration
200 μg/mL
shipped in
wet ice
storage temp.
−20°C
Immunogen
13 amino acids near the carboxy terminus of human IDH1.
Application
Used as a blocking peptide in immunoblotting applications.
Linkage
This peptide blocks the action of antibody SAB3500511.
Disclaimer
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Product No.
Description
Pricing
Storage Class Code
10 - Combustible liquids
Flash Point(F)
Not applicable
Flash Point(C)
Not applicable
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PD n-3 DPA Pathway Regulates Human Monocyte Differentiation and Macrophage Function
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Cell Chemical Biology, 25(6), 749-760 (2018)
You-Yang Qu et al.
Neurochemical research, 40(1), 1-14 (2014-11-05)
Epoxyeicosatrienoic acids (EETs), the cytochrome P450 epoxygenase metabolite of arachidonic acid, have been demonstrated to have neuroprotective effect. Phosphatidylinositol 3-kinase (PI3K)/Akt and ATP-sensitive potassium (KATP) channels are thought to be important factors that mediate neuroprotection. However, little is known about
Soluble epoxide hydrolase inhibitor, 12-(3-adamantan-1-yl-ureido)-dodecanoic acid, represses human aortic smooth muscle cell proliferation and migration by regulating cell death pathways via the mTOR signaling
Li SH, et al.
International Journal of Clinical and Experimental Pathology, 10(8), 8434-8442 (2017)
Aylin Acun et al.
Acta biomaterialia, 94, 372-391 (2019-05-31)
Deaths attributed to ischemic heart disease increased by 41.7% from 1990 to 2013. This is primarily due to an increase in the aged population, however, research on cardiovascular disease (CVD) has been overlooking aging, a well-documented contributor to CVD. The
Jing Li et al.
Frontiers in bioscience : a journal and virtual library, 13, 3480-3487 (2008-05-30)
In stroke-prone spontaneously hypertensive rats (SHRSP) end-organ damage is markedly accelerated by high-salt (HS) intake. Since epoxyeicosatrienoic acids (EETs) possess vasodepressor and natriuretic activities, we examined whether a soluble epoxide hydrolase (sEH) inhibitor, 12-(3-adamantan-1-yl-ureido)-dodecanoic acid (AUDA), to inhibit the metabolism
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