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Key Documents

215A-7

Sigma-Aldrich

Helicobacter pylori Rabbit Polyclonal Antibody

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

rabbit

Quality Level

100
300
500

antibody form

Ig fraction of antiserum

antibody product type

primary antibodies

clone

polyclonal

manufacturer/tradename

Cell Marque®

shipped in

wet ice

storage temp.

2-8°C

Physical form

Solution in Tris Buffer, pH 7.3-7.7, with 1% BSA and <0.1% Sodium Azide

Preparation Note

Download the IFU specific to your product lot and formatNote: This requires a keycode which can be found on your packaging or product label.

Other Notes

Product not available in all regions or countries. Analyte Specific Reagent. Analytical and performance characteristics are not established. To request more information on this product, please contact technical services at 800-665-7284 or email: service@cellmarque.com

Legal Information

Cell Marque is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class

12 - Non Combustible Liquids

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


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M Toulaymat et al.
Archives of pathology & laboratory medicine, 123(9), 778-781 (1999-08-24)
To describe the endoscopic biopsy pathology of Helicobacter pylori gastritis, compare bacterial detection by immunohistochemistry using a specific antibody with the Genta stain, and to compare the relative costs of the 2 techniques. One hundred cases of gastritis identified as
T Shimizu et al.
Helicobacter, 1(4), 197-206 (1996-12-01)
Attempts have been made to develop an accurate method for detecting Helicobacter pylori in histological sections. Biopsy specimens were obtained from the stomachs of 167 patients with gastric ulcer (33), duodenal ulcer (52), gastroduodenal ulcer (15), chronic gastritis (45), and
Nirag C Jhala et al.
American journal of clinical pathology, 119(1), 101-107 (2003-01-11)
It is our hypothesis that if Helicobacter pylori could be demonstrated conclusively to have transgressed the mucosal surface into the lamina propria, this would help explain how H pylori recruits inflammatory cells. We report our immunohistochemical and electron microscopic findings

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