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Sigma-Aldrich

PMS2 (EPR3947) Rabbit Monoclonal Primary Antibody

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.41

biological source

rabbit

Quality Level

antibody form

culture supernatant

antibody product type

primary antibodies

clone

EPR3947, monoclonal

manufacturer/tradename

Cell Marque®

isotype

IgG

shipped in

wet ice

storage temp.

2-8°C

Physical form

Solution in Tris Buffer, pH 7.3-7.7, with 1% BSA and <0.1% Sodium Azide

Preparation Note

Download the IFU specific to your product lot and formatNote: This requires a keycode which can be found on your packaging or product label.

Other Notes

Product not available in all regions or countries. Analyte Specific Reagent in the United States. Analytical and performance characteristics are not established. To request more information on this product, please contact technical services at 800-665-7284 or email: service@cellmarque.com

Legal Information

Cell Marque is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class

12 - Non Combustible Liquids

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable


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Heather Hampel et al.
The New England journal of medicine, 352(18), 1851-1860 (2005-05-06)
Germ-line mutations in the mismatch-repair genes MLH1, MSH2, MSH6, and PMS2 lead to the development of the Lynch syndrome (hereditary nonpolyposis colorectal cancer), conferring a strong susceptibility to cancer. We assessed the frequency of such mutations in patients with colorectal
Adrian Gologan et al.
Clinics in laboratory medicine, 25(1), 179-196 (2005-03-08)
The reference cancers associated with DNA mismatch repair (MMR)deficiency are the adenocarcinomas of patients with hereditary nonpolyposis colorectal cancer, also known as Lynch syndrome. Sporadic gastrointestinal (GI) carcinomas, most commonly colorectal and gastric carcinomas, may also be associated with deficiencies
Yvonne M C Hendriks et al.
Gastroenterology, 130(2), 312-322 (2006-02-14)
The role of the mismatch repair gene PMS2 in hereditary nonpolyposis colorectal carcinoma (HNPCC) is not fully clarified. To date, only 7 different heterozygous truncating PMS2 mutations have been reported in HNPCC-suspected families. Our aim was to further assess the
Kaspar Truninger et al.
Gastroenterology, 128(5), 1160-1171 (2005-05-12)
Germline mutations in the DNA mismatch repair (MMR) genes MSH2, MSH6, or MLH1 predispose to colorectal cancer (CRC) with an autosomal dominant inheritance pattern. The protein encoded by PMS2 is also essential for MMR; however, alterations in this gene have
Janindra Warusavitarne et al.
International journal of colorectal disease, 22(7), 739-748 (2006-11-17)
High-frequency microsatellite instability (MSI-H) is an alternate pathway of colorectal carcinogenesis, which accounts for 15% of all sporadic colorectal cancers. These tumours arise from mutations in the DNA mismatch repair system and thus have different responses to chemotherapeutic agents compared

Articles

Colorectal cancer is a common occurrence among inhabitants of most Western countries, second only to carcinoma of the lung.

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